Medical Disclaimer: This is educational content only, not medical advice. Consult a licensed healthcare provider for diagnosis/treatment. Information based on sources like WHO/CDC guidelines (last reviewed: 2026-02-13).
Medical Disclaimer: This is educational content only, not medical advice. Consult a licensed healthcare provider for diagnosis/treatment. Information based on sources like WHO/CDC guidelines (last reviewed: 2026-02-13).
Case scenarios integrate anatomy with clinical presentation, helping students apply theoretical knowledge to real-life neurological problems, which is essential for medical entrance and university exams.
Spinothalamic tract lesions present as contralateral loss of pain and temperature sensation starting one to two segments below the level of the lesion, with preserved proprioception.
UMN lesions cause spasticity, hyperreflexia, clonus, and positive Babinski sign, while LMN lesions produce flaccid paralysis, muscle wasting, fasciculations, and absent reflexes.
Syringomyelia damages the anterior white commissure where spinothalamic fibers cross, leading to bilateral segmental loss of pain and temperature over shoulders and upper limbs.
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